Lose more weight by properly matching
diet and exercise to your personal genotypes*. What's the right percentage of carbs, fat and protein for your diet? Should you exercise at high intensity, or can you get by with a moderate workout?
Your test results may tell you the personalized answers you need to achieve your healthy weight loss goals.
A simple swab is all it takes. Take the test in the comfort of your home -
it's quick and easy, just a swab of your cheek. Then simply drop your swab in the mail to our 100% confidential CLIA-certified lab.
You'll receive a personalized, easy to understand Test Report and Results Booklet recommending a diet and exercise plan that may work better
with your genetic makeup to support your weight management goals**.
We now know that most heart attacks are not simply a gradual build-up of a blockage in an artery - like a blocked drain -
but the result of a much more active complex process, driven by a long-lasting inflammatory response.
Inflammation is essential to life, defending the body against infection and helping coordinate the repair of damaged tissues and organs.
But when these processes persist for years, they can create fundamental changes to the tissue, such as what occurs in the artery wall
of a person that suffers a heart attack.
The interleukin-1 (IL-1) family of cytokines plays a key role in coordinating and regulating inflammation. Importantly, subtle differences in two IL-1 genes,
IL1A and IL1B, are associated with increased risk for earlier or more severe cardiovascular disease and heart attacks.
The Heart Health genetic test identifies individuals who test positive for these IL-1 genetic patterns. These genetic patterns increase cardiovascular risk
by a similar amount to other, better known risk factors - such as smoking and high LDL cholesterol - which can increase your overall risk.
Knowing your IL-1 genotype allows you take control of your heart health by enacting lifestyle changes to manage your specific risk factors.
We are what we eat, and despite there being no evidence that eating carrots will improve your eyesight (as your mother probably tried to convince you),
there is bountiful evidence that eating a balanced diet is important for our overall health and wellness. Just how careful we need to be about this
is influenced by our genetic make-up.
To maintain good health, we need vitamins and nutrients to supplement the energy obtained from fats, carbohydrates and proteins.
These are found in many of the healthy foods available to us, and if we are not getting enough in our diet, we can make up the difference
with the right vitamins and supplements. Just how much is 'enough' can vary due to genetic variations in some of the proteins that regulate
how our bodies use these nutrients; for some of us a 'standard' dietary intake can leave us at risk of developing certain diseases.
Individuals with certain variations in two of the genes that control B vitamin metabolism, called MTHFR and TCN2, do not metabolize B vitamins
as effectively as the rest of us. If this is not accounted for by increasing their dietary intake of B vitamins, they may be at an increased risk
of developing heart disease, and of giving birth to children with spinal cord defects.
Osteoporosis, the most common age-related bone disease, results in a decrease in the strength of the bone that leaves the affected individual
more susceptible to fractures. According to the National Institute of Health, 10 million Americans suffer from the disease and another 34 million
have low bone mass, placing them at increased risk for the disease.
Although osteoporosis occurs in both men and women, it begins earlier and progresses more rapidly in women after menopause. The consequences
of osteoporosis can be both physical and financial. Hip and vertebral fractures, which are commonly associated with osteoporosis,
have a profound impact on quality of life.
We have conducted research projects with major osteoporosis centers. Results of these studies have indicated that a number of small variations
in the IL-1 gene cluster, referred to as polymorphisms, are associated with a more rapid rate of bone loss and an increased risk
of vertebral fracture in post-menopausal Caucasian women.